Gene description for Atp6v1h
Gene name ATPase, H+ transporting, lysosomal V1 subunit H
Gene symbol Atp6v1h
Other names/aliases 0710001F19Rik
AU022349
CGI-11
SFD
SFDalpha
SFDbeta
VMA13
Species Mus musculus
 Database cross references - Atp6v1h
ExoCarta ExoCarta_108664
Entrez Gene 108664
UniProt Q8BVE3  
 Atp6v1h identified in exosomes derived from the following tissue/cell type
Fibroblasts 23260141    
 Gene ontology annotations for Atp6v1h
Molecular Function
    hydrolase activity, acting on acid anhydrides, catalyzing transmembrane movement of substances GO:0016820 IEA
    proton-transporting ATPase activity, rotational mechanism GO:0046961 IEA
Biological Process
    endocytosis GO:0006897 ISO
    transport GO:0006810 IEA
    ATP hydrolysis coupled proton transport GO:0015991 IEA
    proton transport GO:0015992 IEA
    ion transport GO:0006811 IEA
Subcellular Localization
    vacuolar proton-transporting V-type ATPase, V1 domain GO:0000221 IEA
    lysosomal membrane GO:0005765 ISO
    extracellular exosome GO:0070062 ISO
 Experiment description of studies that identified Atp6v1h in exosomes
1
Experiment ID 210
ISEV standards
EM
EV Biophysical techniques
EV Cytosolic markers
CD81|FLOT1
EV Membrane markers
EV Negative markers
EV Particle analysis
Identified molecule protein
Identification method Mass spectrometry
PubMed ID 23260141    
Organism Mus musculus
Experiment description Exosomes Mediate Stromal Mobilization of Autocrine Wnt-PCP Signaling in Breast Cancer Cell Migration.
Authors Luga V, Zhang L, Viloria-Petit AM, Ogunjimi AA, Inanlou MR, Chiu E, Buchanan M, Hosein AN, Basik M, Wrana JL.
Journal name Cell
Publication year 2012
Sample Fibroblasts
Sample name Normal-Fibroblasts (L cells)
Isolation/purification methods Differential centrifugation
Ultracentrifugation
Flotation density -
Molecules identified in the study Protein
Methods used in the study Mass spectrometry
 Protein-protein interactions for Atp6v1h
  Protein Interactor ExoCarta ID Identification method PubMed Species
No interactions are found.
 Pathways in which Atp6v1h is involved
PathwayEvidenceSource
Insulin receptor recycling IEA Reactome
Ion channel transport IEA Reactome
Transferrin endocytosis and recycling IEA Reactome





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